One simple diagnostic method to determine foot insensitivity is a nanofilament test or sending a weak electric shock to the patient to detect their response. However, accuracy is an issue due to controlling neurostimulation in the patient. We have developed a device that can detect neuro signals using an electric wavelength between 3-100Hz. We utilize TGAM, which is typically used to detect EEG signals. The device is connected to three different parts located near the Achilles tendon. Synapse signals were collected from 5 normal and diabetic subjects (fasting glucose level over 180mg/dl) by lifting the foot every 3 seconds for 1 minute, using electrodes coated with AgCl. Analog data from the subjects were converted into decimal data and plotted. From the 1-minute response, diabetes patients generated an average of 18.21 signaling spots, whereas normal subjects generated an average of 26.49 signaling spots. Additionally, the total signal created by normal subjects is 633.42% greater than that of diabetes patients. Based on the results, we can conclude that impaired foot movement occurs in diabetes patients due to high glucose levels, which may delay muscle movement by transmitting smaller signal numbers. Furthermore, the signal stimulation itself is also less than normal. Therefore, the progression of diabetic foot may decrease patient movement due to function loss, resulting from reduced signal activity and poor signal transmission.
W. Kim: None. M.S. Park: None.
