CTLA4 gene haplotypes cannot protect from IDDM in the presence of high-risk HLA DQ8 or DQ2 alleles in German families.

Donner, H; Seidl, C; Braun, J; Siegmund, T; Herwig, J; Seifried, E; Usadel, K H; Badenhoop, K

doi:10.2337/diabetes.47.7.1158

H Donner;

H Donner

Medical Department I, Center for Internal Medicine, Johann Wolfgang Goethe University Hospital, Frankfurt/Main, Germany.

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C Seidl;

C Seidl

Medical Department I, Center for Internal Medicine, Johann Wolfgang Goethe University Hospital, Frankfurt/Main, Germany.

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J Braun;

J Braun

Medical Department I, Center for Internal Medicine, Johann Wolfgang Goethe University Hospital, Frankfurt/Main, Germany.

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T Siegmund;

T Siegmund

Medical Department I, Center for Internal Medicine, Johann Wolfgang Goethe University Hospital, Frankfurt/Main, Germany.

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J Herwig;

J Herwig

Medical Department I, Center for Internal Medicine, Johann Wolfgang Goethe University Hospital, Frankfurt/Main, Germany.

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E Seifried;

E Seifried

Medical Department I, Center for Internal Medicine, Johann Wolfgang Goethe University Hospital, Frankfurt/Main, Germany.

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K H Usadel;

K H Usadel

Medical Department I, Center for Internal Medicine, Johann Wolfgang Goethe University Hospital, Frankfurt/Main, Germany.

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K Badenhoop

Medical Department I, Center for Internal Medicine, Johann Wolfgang Goethe University Hospital, Frankfurt/Main, Germany.

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CTLA4 gene haplotypes cannot protect from IDDM in the presence of high-risk HLA DQ8 or DQ2 alleles in German families.

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CTLA4 gene haplotypes cannot protect from IDDM in the presence of high-risk HLA DQ8 or DQ2 alleles in German families.

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