Caramori ML, Kim Y, Huang C, Fish AJ, Rich SS, Miller ME, Russell G, Mauer M: Cellular basis of diabetic nephropathy. 1. Study design and renal structural–functional relationships in patients with long-standing type 1 diabetes. Diabetes 51:506–513, 2002

In the above article, the symbols in the legends for Figs. 3, 4, and 5 were given incorrectly. The correct legends are shown here.

FIG. 3.

Correlation between mesangial fraction volume [Vv(Mes/glom)] and AER in 124 type 1 diabetic patients. ♦, normoalbuminuric patients; □, microalbuminuric patients; ▵, proteinuric patients. r = 0.75, P < 0.001.

FIG. 3.

Correlation between mesangial fraction volume [Vv(Mes/glom)] and AER in 124 type 1 diabetic patients. ♦, normoalbuminuric patients; □, microalbuminuric patients; ▵, proteinuric patients. r = 0.75, P < 0.001.

Close modal
FIG. 4.

Correlation between GBM width and AER in 124 type 1 diabetic patients. ♦, normoalbuminuric patients; □, microalbuminuric patients; ▵, proteinuric patients. r = 0.63, P < 0.001.

FIG. 4.

Correlation between GBM width and AER in 124 type 1 diabetic patients. ♦, normoalbuminuric patients; □, microalbuminuric patients; ▵, proteinuric patients. r = 0.63, P < 0.001.

Close modal
FIG. 5.

Correlation between surface density of peripheral GBM per glomerulus [Sv(PGBM/glom)] and GFR in 125 type 1 diabetic patients. ♦, normoalbuminuric patients; □, microalbuminuric patients; ▵, proteinuric patients. r = 0.48, P < 0.001.

FIG. 5.

Correlation between surface density of peripheral GBM per glomerulus [Sv(PGBM/glom)] and GFR in 125 type 1 diabetic patients. ♦, normoalbuminuric patients; □, microalbuminuric patients; ▵, proteinuric patients. r = 0.48, P < 0.001.

Close modal