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Activators of peroxisome proliferator-activated receptor-alpha induce the expression of the uncoupling protein-3 gene in skeletal muscle: a potential mechanism for the lipid intake-dependent activation of uncoupling protein-3 gene expression at birth.
Diabetes June 1999, Vol.48, 1217-1222. doi:https://doi.org/10.2337/diabetes.48.6.1217
Transfection of human pancreatic islets with an anti-apoptotic gene (bcl-2) protects beta-cells from cytokine-induced destruction.
A Rabinovitch; W Suarez-Pinzon; K Strynadka; Q Ju; D Edelstein; M Brownlee; G S Korbutt; R V Rajotte
Diabetes June 1999, Vol.48, 1223-1229. doi:https://doi.org/10.2337/diabetes.48.6.1223
Impaired beta-cell functions induced by chronic exposure of cultured human pancreatic islets to high glucose.
Diabetes June 1999, Vol.48, 1230-1236. doi:https://doi.org/10.2337/diabetes.48.6.1230
Impaired free fatty acid uptake in skeletal muscle but not in myocardium in patients with impaired glucose tolerance: studies with PET and 14(R,S)-[18F]fluoro-6-thia-heptadecanoic acid.
A K Turpeinen; T O Takala; P Nuutila; T Axelin; M Luotolahti; M Haaparanta; J Bergman; H Hämäläinen; H Iida; M Mäki; M I Uusitupa; J Knuuti
Diabetes June 1999, Vol.48, 1245-1250. doi:https://doi.org/10.2337/diabetes.48.6.1245
Modifications of citric acid cycle activity and gluconeogenesis in streptozotocin-induced diabetes and effects of metformin.
Diabetes June 1999, Vol.48, 1251-1257. doi:https://doi.org/10.2337/diabetes.48.6.1251
Triglyceride-induced diabetes associated with familial lipoprotein lipase deficiency.
G Mingrone; F L Henriksen; A V Greco; L N Krogh; E Capristo; A Gastaldelli; M Castagneto; E Ferrannini; G Gasbarrini; H Beck-Nielsen
Diabetes June 1999, Vol.48, 1258-1263. doi:https://doi.org/10.2337/diabetes.48.6.1258
Acute intravenous leptin infusion increases glucose turnover but not skeletal muscle glucose uptake in ob/ob mice.
Diabetes June 1999, Vol.48, 1264-1269. doi:https://doi.org/10.2337/diabetes.48.6.1264
Free fatty acid-induced insulin resistance is associated with activation of protein kinase C theta and alterations in the insulin signaling cascade.
M E Griffin; M J Marcucci; G W Cline; K Bell; N Barucci; D Lee; L J Goodyear; E W Kraegen; M F White; G I Shulman
Diabetes June 1999, Vol.48, 1270-1274. doi:https://doi.org/10.2337/diabetes.48.6.1270
Low plasma leptin levels contribute to diabetic hyperphagia in rats.
Diabetes June 1999, Vol.48, 1275-1280. doi:https://doi.org/10.2337/diabetes.48.6.1275
Effects of aggressive cholesterol lowering and low-dose anticoagulation on clinical and angiographic outcomes in patients with diabetes: the Post Coronary Artery Bypass Graft Trial.
B J Hoogwerf; A Waness; M Cressman; J Canner; L Campeau; M Domanski; N Geller; A Herd; A Hickey; D B Hunninghake; G L Knatterud; C White
Diabetes June 1999, Vol.48, 1289-1294. doi:https://doi.org/10.2337/diabetes.48.6.1289
Endothelium-dependent vasodilatation, plasma markers of endothelial function, and adrenergic vasoconstrictor responses in type 1 diabetes under near-normoglycemic conditions.
F C Huvers; P W De Leeuw; A J Houben; C H De Haan; K Hamulyak; H Schouten; B H Wolffenbuttel; N C Schaper
Diabetes June 1999, Vol.48, 1300-1307. doi:https://doi.org/10.2337/diabetes.48.6.1300
Dietary glycotoxins: inhibition of reactive products by aminoguanidine facilitates renal clearance and reduces tissue sequestration.
Diabetes June 1999, Vol.48, 1308-1315. doi:https://doi.org/10.2337/diabetes.48.6.1308
Protein kinase C activity is acutely regulated by plasma glucose concentration in human monocytes in vivo.
Diabetes June 1999, Vol.48, 1316-1322. doi:https://doi.org/10.2337/diabetes.48.6.1316
Human diabetes is associated with hyperreactivity of vascular smooth muscle cells due to altered subcellular Ca2+ distribution.
E Fleischhacker; V E Esenabhalu; M Spitaler; S Holzmann; F Skrabal; B Koidl; G M Kostner; W F Graier
Diabetes June 1999, Vol.48, 1323-1330. doi:https://doi.org/10.2337/diabetes.48.6.1323
Identification of the high-affinity tolbutamide site on the SUR1 subunit of the K(ATP) channel.
Diabetes June 1999, Vol.48, 1341-1347. doi:https://doi.org/10.2337/diabetes.48.6.1341