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Journal Articles

Loss of β-cell K_ATP reduces Ca²⁺ sensitivity of insulin secretion and Trpm5 expression

Nathaniel W. York, Zihan Yan, Anna B. Osipovich, Abbie Tate, Sumit Patel, David W. Piston, Mark A. Magnuson, Maria S. Remedi, Colin G. Nichols

Diabetes db240650

https://doi.org/10.2337/db24-0650

Published: 12 December 2024

View article titled, Loss of β-cell KATP reduces Ca2+ sensitivity of insulin secretion and Trpm5 expression

Journal Articles

Glucokinase Inhibition: A Novel Treatment for Diabetes?

Maria S. Remedi, Colin G. Nichols

Diabetes 2023;72(2):170–174

https://doi.org/10.2337/db22-0731

Published: 20 January 2023

View article titled, Glucokinase Inhibition: A Novel Treatment for Diabetes?

Journal Articles

Genetic Reduction of Glucose Metabolism Preserves Functional β-Cell Mass in K_ATP-Induced Neonatal Diabetes

Zihan Yan, Manuela Fortunato, Zeenat A. Shyr, Amy L. Clark, Matt Fuess, Colin G. Nichols, Maria S. Remedi

Diabetes 2022;71(6):1233–1245

https://doi.org/10.2337/db21-0992

Published: 16 March 2022

View article titled, Genetic Reduction of Glucose Metabolism Preserves Functional β-Cell Mass in KATP-Induced Neonatal Diabetes

Journal Articles

ATP-Sensitive Potassium Channels in Hyperinsulinism and Type 2 Diabetes: Inconvenient Paradox or New Paradigm?

Colin G. Nichols, Nathaniel W. York, Maria S. Remedi

Diabetes 2022;71(3):367–375

https://doi.org/10.2337/db21-0755

Published: 23 February 2022

View article titled, ATP-Sensitive Potassium Channels in Hyperinsulinism and Type 2 Diabetes: Inconvenient Paradox or New Paradigm?

Journal Articles

ABCC8 R1420H Loss-of-Function Variant in a Southwest American Indian Community: Association With Increased Birth Weight and Doubled Risk of Type 2 Diabetes

Leslie J. Baier, Yunhua Li Muller, Maria Sara Remedi, Michael Traurig, Paolo Piaggi, Gregory Wiessner, Ke Huang, Alyssa Stacy, Sayuko Kobes, Jonathan Krakoff, Peter H. Bennett, Robert G. Nelson, William C. Knowler, Robert L. Hanson, Colin G. Nichols, Clifton Bogardus

Diabetes 2015;64(12):4322–4332

https://doi.org/10.2337/db15-0459

Published: 05 August 2015

View article titled, ABCC8 R1420H Loss-of-Function Variant in a Southwest American Indian Community: Association With Increased Birth Weight and Doubled Risk of Type 2 Diabetes

Includes: Supplementary data

Journal Articles

Acute Sulfonylurea Therapy at Disease Onset Can Cause Permanent Remission of K_ATP-Induced Diabetes

Maria Sara Remedi, Sophia E. Agapova, Arpita K. Vyas, Paul W. Hruz, Colin G. Nichols

Diabetes 2011;60(10):2515–2522

https://doi.org/10.2337/db11-0538

Published: 16 September 2011

View article titled, Acute Sulfonylurea Therapy at Disease Onset Can Cause Permanent Remission of KATP-Induced Diabetes

Journal Articles

Congenital Hyperinsulinism and Glucose Hypersensitivity in Homozygous and Heterozygous Carriers of Kir6.2 (KCNJ11) Mutation V290M Mutation: K_ATP Channel Inactivation Mechanism and Clinical Management

Karen J. Loechner, Alejandro Akrouh, Harley T. Kurata, Carlo Dionisi-Vici, Arianna Maiorana, Milena Pizzoferro, Vittoria Rufini, Jean de Ville de Goyet, Carlo Colombo, Fabrizio Barbetti, Joseph C. Koster, Colin G. Nichols

Diabetes 2011;60(1):209–217

https://doi.org/10.2337/db10-0731

Published: 27 October 2010

View article titled, Congenital Hyperinsulinism and Glucose Hypersensitivity in Homozygous and Heterozygous Carriers of Kir6.2 (KCNJ11) Mutation V290M Mutation: <subtitle>KATP Channel Inactivation Mechanism and Clinical Management</subtitle>

Journal Articles

Kir6.2 Variant E23K Increases ATP-Sensitive K⁺ Channel Activity and Is Associated With Impaired Insulin Release and Enhanced Insulin Sensitivity in Adults With Normal Glucose Tolerance

Dennis T. Villareal, Joseph C. Koster, Heather Robertson, Alejandro Akrouh, Kazuaki Miyake, Graeme I. Bell, Bruce W. Patterson, Colin G. Nichols, Kenneth S. Polonsky

Diabetes 2009;58(8):1869–1878

https://doi.org/10.2337/db09-0025

Published: 02 June 2009

View article titled, Kir6.2 Variant E23K Increases ATP-Sensitive K+ Channel Activity and Is Associated With Impaired Insulin Release and Enhanced Insulin Sensitivity in Adults With Normal Glucose Tolerance

Includes: Supplementary data

Journal Articles

A Mutation in the TMD0-L0 Region of Sulfonylurea Receptor-1 (L225P) Causes Permanent Neonatal Diabetes Mellitus (PNDM)

Ricard Masia, Diva D. De Leon, Courtney MacMullen, Heather McKnight, Charles A. Stanley, Colin G. Nichols

Diabetes 2007;56(5):1357–1362

https://doi.org/10.2337/db06-1746

Published: 01 May 2007

View article titled, A Mutation in the TMD0-L0 Region of Sulfonylurea Receptor-1 (L225P) Causes Permanent Neonatal Diabetes Mellitus (PNDM)

Journal Articles

An ATP-Binding Mutation (G334D) in KCNJ11 Is Associated With a Sulfonylurea-Insensitive Form of Developmental Delay, Epilepsy, and Neonatal Diabetes

Ricard Masia, Joseph C. Koster, Stefano Tumini, Francesco Chiarelli, Carlo Colombo, Colin G. Nichols, Fabrizio Barbetti

Diabetes 2007;56(2):328–336

https://doi.org/10.2337/db06-1275

Published: 01 February 2007

View article titled, An ATP-Binding Mutation (G334D) in KCNJ11 Is Associated With a Sulfonylurea-Insensitive Form of Developmental Delay, Epilepsy, and Neonatal Diabetes

Journal Articles

Expression of ATP-Insensitive K_ATP Channels in Pancreatic β-Cells Underlies a Spectrum of Diabetic Phenotypes

Joseph C. Koster, Maria S. Remedi, Ricard Masia, Brian Patton, Ailing Tong, Colin G. Nichols

Diabetes 2006;55(11):2957–2964

https://doi.org/10.2337/db06-0732

Published: 01 November 2006

View article titled, Expression of ATP-Insensitive KATP Channels in Pancreatic β-Cells Underlies a Spectrum of Diabetic Phenotypes

Journal Articles

Diabetes and Insulin Secretion: The ATP-Sensitive K⁺ Channel (K_ATP) Connection

Joseph C. Koster, M. Alan Permutt, Colin G. Nichols

Diabetes 2005;54(11):3065–3072

https://doi.org/10.2337/diabetes.54.11.3065

Published: 01 November 2005

View article titled, Diabetes and Insulin Secretion: <subtitle>The ATP-Sensitive K+ Channel (KATP) Connection</subtitle>

Journal Articles

ATP-Sensitive K⁺ Channel Signaling in Glucokinase-Deficient Diabetes

Maria S. Remedi, Joseph C. Koster, Brian L. Patton, Colin G. Nichols

Diabetes 2005;54(10):2925–2931

https://doi.org/10.2337/diabetes.54.10.2925

Published: 01 October 2005

View article titled, ATP-Sensitive K+ Channel Signaling in Glucokinase-Deficient Diabetes

Journal Articles

ATP and Sulfonylurea Sensitivity of Mutant ATP-Sensitive K⁺ Channels in Neonatal Diabetes: Implications for Pharmacogenomic Therapy

Joseph C. Koster, Maria S. Remedi, Crystal Dao, Colin G. Nichols

Diabetes 2005;54(9):2645–2654

https://doi.org/10.2337/diabetes.54.9.2645

Published: 01 September 2005

View article titled, ATP and Sulfonylurea Sensitivity of Mutant ATP-Sensitive K+ Channels in Neonatal Diabetes: <subtitle>Implications for Pharmacogenomic Therapy</subtitle>

Journal Articles

Diet-Induced Glucose Intolerance in Mice With Decreased β-Cell ATP-Sensitive K⁺ Channels

Maria S. Remedi, Joseph. C. Koster, Kamelia Markova, Susumu Seino, Takashi Miki, Brian L. Patton, Michael L. McDaniel, Colin G. Nichols

Diabetes 2004;53(12):3159–3167

https://doi.org/10.2337/diabetes.53.12.3159

Published: 01 December 2004

View article titled, Diet-Induced Glucose Intolerance in Mice With Decreased β-Cell ATP-Sensitive K+ Channels

Journal Articles

HIV Protease Inhibitors Acutely Impair Glucose-Stimulated Insulin Release

Joseph C. Koster, Maria S. Remedi, Haijun Qiu, Colin G. Nichols, Paul W. Hruz

Diabetes 2003;52(7):1695–1700

https://doi.org/10.2337/diabetes.52.7.1695

Published: 01 July 2003

View article titled, HIV Protease Inhibitors Acutely Impair Glucose-Stimulated Insulin Release

NARROW

Loss of β-cell K_ATP reduces Ca²⁺ sensitivity of insulin secretion and Trpm5 expression

Glucokinase Inhibition: A Novel Treatment for Diabetes?

Genetic Reduction of Glucose Metabolism Preserves Functional β-Cell Mass in K_ATP-Induced Neonatal Diabetes

ATP-Sensitive Potassium Channels in Hyperinsulinism and Type 2 Diabetes: Inconvenient Paradox or New Paradigm?

ABCC8 R1420H Loss-of-Function Variant in a Southwest American Indian Community: Association With Increased Birth Weight and Doubled Risk of Type 2 Diabetes

Acute Sulfonylurea Therapy at Disease Onset Can Cause Permanent Remission of K_ATP-Induced Diabetes

Congenital Hyperinsulinism and Glucose Hypersensitivity in Homozygous and Heterozygous Carriers of Kir6.2 (KCNJ11) Mutation V290M Mutation: K_ATP Channel Inactivation Mechanism and Clinical Management

Kir6.2 Variant E23K Increases ATP-Sensitive K⁺ Channel Activity and Is Associated With Impaired Insulin Release and Enhanced Insulin Sensitivity in Adults With Normal Glucose Tolerance

A Mutation in the TMD0-L0 Region of Sulfonylurea Receptor-1 (L225P) Causes Permanent Neonatal Diabetes Mellitus (PNDM)

An ATP-Binding Mutation (G334D) in KCNJ11 Is Associated With a Sulfonylurea-Insensitive Form of Developmental Delay, Epilepsy, and Neonatal Diabetes

Expression of ATP-Insensitive K_ATP Channels in Pancreatic β-Cells Underlies a Spectrum of Diabetic Phenotypes

Diabetes and Insulin Secretion: The ATP-Sensitive K⁺ Channel (K_ATP) Connection

ATP-Sensitive K⁺ Channel Signaling in Glucokinase-Deficient Diabetes

ATP and Sulfonylurea Sensitivity of Mutant ATP-Sensitive K⁺ Channels in Neonatal Diabetes: Implications for Pharmacogenomic Therapy

Diet-Induced Glucose Intolerance in Mice With Decreased β-Cell ATP-Sensitive K⁺ Channels

HIV Protease Inhibitors Acutely Impair Glucose-Stimulated Insulin Release

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