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Seokwon Jo
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Meeting Abstracts

205-OR: Nutrient Sensor OGT Regulates Glucose Homeostasis through ß-Cell Autophagy via mTORC1 Pathway

SEOKWON JO, EMILYN ALEJANDRO
Journal: Diabetes
Diabetes 2022;71(Supplement_1):205-OR
https://doi.org/10.2337/db22-205-OR
Published: 01 June 2022
View article titled, 205-OR: Nutrient Sensor OGT Regulates Glucose Homeostasis through ß-Cell Autophagy via mTORC1 Pathway
Journal Articles

Imeglimin to the Rescue: Enhanced CHOP/GADD34/eIF2α Signaling Axis Promotes β-Cell Survival

Seokwon Jo, Emilyn U. Alejandro
Journal: Diabetes
Diabetes 2022;71(3):376–378
https://doi.org/10.2337/dbi21-0035
Published: 23 February 2022
View article titled, Imeglimin to the Rescue: Enhanced CHOP/GADD34/eIF2α Signaling Axis Promotes β-Cell Survival
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Journal Articles

OGT Regulates Mitochondrial Biogenesis and Function via Diabetes Susceptibility Gene Pdx1

Ramkumar Mohan, Seokwon Jo, Amber Lockridge, Deborah A. Ferrington, Kevin Murray, Arthur Eschenlauer, Ernesto Bernal-Mizrachi, Yoshio Fujitani, Emilyn U. Alejandro
Journal: Diabetes
Diabetes 2021;70(11):2608–2625
https://doi.org/10.2337/db21-0468
Published: 30 August 2021
View article titled, OGT Regulates Mitochondrial Biogenesis and Function via Diabetes Susceptibility Gene Pdx1
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Journal Articles

Translational Factor eIF4G1 Regulates Glucose Homeostasis and Pancreatic β-Cell Function

Seokwon Jo, Amber Lockridge, Ramkumar Mohan, Nicholas Esch, Regina Schlichting, Neha Panigrahy, Ahmad Essawy, Eric Gustafson, Emilyn U. Alejandro
Journal: Diabetes
Diabetes 2021;70(1):155–170
https://doi.org/10.2337/db20-0057
Published: 28 October 2020
View article titled, Translational Factor eIF4G1 Regulates Glucose Homeostasis and Pancreatic β-Cell Function
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Meeting Abstracts

Newly Revealed Role for OGT in Obesity-Precipitated ß-Cell Hyperinsulinemia

AMBER D. LOCKRIDGE, SEOKWON JO, NIKLAS E. DAMBERG, RAMKUMAR MOHAN, EMILYN ALEJANDRO
Journal: Diabetes
Diabetes 2018;67(Supplement_1):2161-P
https://doi.org/10.2337/db18-2161-P
Published: 01 July 2018
View article titled, Newly Revealed Role for OGT in Obesity-Precipitated ß-Cell Hyperinsulinemia
Meeting Abstracts

Hyperproinsulinemia in OGlcNAc Transferase Deficient Mice Is Associated with Loss of CPE and eIF4G1, but ß-Cell Deletion of eIF4G1 Induces Glucose Intolerance Independent of CPE Dysregulation

SEOKWON JO, AMBER D. LOCKRIDGE, EMILYN ALEJANDRO
Journal: Diabetes
Diabetes 2018;67(Supplement_1):2167-P
https://doi.org/10.2337/db18-2167-P
Published: 01 July 2018
View article titled, Hyperproinsulinemia in OGlcNAc Transferase Deficient Mice Is Associated with Loss of CPE and eIF4G1, but ß-Cell Deletion of eIF4G1 Induces Glucose Intolerance Independent of CPE Dysregulation
Meeting Abstracts

Deletion of OGlcNAc Transferase in Pancreatic Progenitors Causes Partial Pancreas Agenesis and Diabetes Independent of p53 in Mice

EMILYN ALEJANDRO, DANIEL BAUMANN, SEOKWON JO, BRIAN AKHAPHONG, AMBER D. LOCKRIDGE, RAMKUMAR MOHAN
Journal: Diabetes
Diabetes 2018;67(Supplement_1):195-OR
https://doi.org/10.2337/db18-195-OR
Published: 01 July 2018
View article titled, Deletion of OGlcNAc Transferase in Pancreatic Progenitors Causes Partial Pancreas Agenesis and Diabetes Independent of p53 in Mice