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Zeenat A. Shyr
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Meeting Abstracts

318-OR: Intermittent Fasting Improves ß-cell Function in Nonobese KATP-Induced Neonatal Diabetes through Reduction of Cellular Stress and Enhanced Autophagy

ESMERALDA CASTELBLANCO, ZEENAT A. SHYR, ZIHAN YAN, ABHINAV DIWAN, MARIA S. REMEDI
Journal: Diabetes
Diabetes 2022;71(Supplement_1):318-OR
https://doi.org/10.2337/db22-318-OR
Published: 01 June 2022
View articletitled, 318-OR: Intermittent Fasting Improves ß-cell Function in Nonobese KATP-Induced Neonatal Diabetes through Reduction of Cellular Stress and Enhanced Autophagy
Journal Articles

Genetic Reduction of Glucose Metabolism Preserves Functional β-Cell Mass in KATP-Induced Neonatal Diabetes

Zihan Yan, Manuela Fortunato, Zeenat A. Shyr, Amy L. Clark, Matt Fuess, Colin G. Nichols, Maria S. Remedi
Journal: Diabetes
Diabetes 2022;71(6):1233–1245
https://doi.org/10.2337/db21-0992
Published: 16 March 2022
View articletitled, Genetic Reduction of Glucose Metabolism Preserves Functional β-Cell Mass in KATP-Induced Neonatal Diabetes
Open the PDF for Genetic Reduction of Glucose Metabolism Preserves Functional β-Cell Mass in KATP-Induced Neonatal Diabetes in another window
Meeting Abstracts

1789-P: Inactivation of Acyl-Protein Thioesterase 1, a Major Mediator of Palmitoylation Cycling, Promotes Insulin Secretion and ß-Cell Failure

GUIFANG DONG, XIAOCHAO WEI, LI YIN, CHU FENG, SANGEETA ADAK, ZEENAT A. SHYR, QIANG ZHANG, SHUNTARO MORIKAWA, RIE ASADA, FUMIHIKO URANO, MARIA S. REMEDI, CLAY F. SEMENKOVICH
Journal: Diabetes
Diabetes 2020;69(Supplement_1):1789-P
https://doi.org/10.2337/db20-1789-P
Published: 01 June 2020
View articletitled, 1789-P: Inactivation of Acyl-Protein Thioesterase 1, a Major Mediator of Palmitoylation Cycling, Promotes Insulin Secretion and ß-Cell Failure
Meeting Abstracts

334-LB: Dapagliflozin, a SGLT2 Inhibitor, Therapy Improves ß-Cell Function in Neonatal Diabetes by Reducing Oxidative and ER Stress, and Loss of ß-Cell Identity

ZIHAN YAN, ZEENAT A. SHYR, MARIA S. REMEDI
Journal: Diabetes
Diabetes 2019;68(Supplement_1):334-LB
https://doi.org/10.2337/db19-334-LB
Published: 01 June 2019
View articletitled, 334-LB: Dapagliflozin, a SGLT2 Inhibitor, Therapy Improves ß-Cell Function in Neonatal Diabetes by Reducing Oxidative and ER Stress, and Loss of ß-Cell Identity
Meeting Abstracts

Mechanisms Underlying the Progressive Loss of Beta-Cell Function and Mass in Neonatal Diabetes Are Reverted by Dapagliflozin Therapy

ZEENAT A. SHYR, ZIHAN YAN, ALESSANDRO USTIONE, MARIA S. REMEDI
Journal: Diabetes
Diabetes 2018;67(Supplement_1):2158-P
https://doi.org/10.2337/db18-2158-P
Published: 01 July 2018
View articletitled, Mechanisms Underlying the Progressive Loss of Beta-Cell Function and Mass in Neonatal Diabetes Are Reverted by Dapagliflozin Therapy
Meeting Abstracts

Beta-Cell Glucotoxicity in KATP-Induced Diabetes Is Alleviated by Genetic Reduction of Glucose Metabolism

MANUELA FORTUNATO, ZIHAN YAN, ZEENAT A. SHYR, HANNAH E. CONWAY, MARIA S. REMEDI
Journal: Diabetes
Diabetes 2018;67(Supplement_1):327-OR
https://doi.org/10.2337/db18-327-OR
Published: 01 July 2018
View articletitled, Beta-Cell Glucotoxicity in KATP-Induced Diabetes Is Alleviated by Genetic Reduction of Glucose Metabolism