Page 1 | Search Results | Diabetes | American Diabetes Association

1-20 of 67

Sort by

Sort Order Select

Journal Articles

Loss of ATP-Sensitive Potassium Channel Expression and Function in the Nervous System Decreases Opioid Sensitivity in a High-Fat Diet–Fed Mouse Model of Diet-Induced Obesity

Cole Fisher, Kayla Johnson, Madelyn Moore, Amir Sadrati, Jody L. Janecek, Melanie L. Graham, Amanda H. Klein

Diabetes 2024;73(8):1244–1254

https://doi.org/10.2337/db23-1030

Published: 22 May 2024

View article titled, Loss of ATP-Sensitive Potassium Channel Expression and Function in the Nervous System Decreases Opioid Sensitivity in a High-Fat Diet–Fed Mouse Model of Diet-Induced Obesity

Meeting Abstracts

1749-P: Influence of the Biomechanical Environment on Islet Function and Maturity

KELLY VAZQUEZ, RICHARD K. BENNINGER

Diabetes 2023;72(Supplement_1):1749-P

https://doi.org/10.2337/db23-1749-P

Published: 20 June 2023

View article titled, 1749-P: Influence of the Biomechanical Environment on Islet Function and Maturity

Meeting Abstracts

287-OR: An ABCC8 R1420H Loss-of-Function Variant Affects Insulin Secretory Function and Microvascular Disease in American Indians

ELSA VAZQUEZ ARREOLA, WILLIAM C. KNOWLER, LESLIE BAIER, ROBERT L. HANSON

Diabetes 2023;72(Supplement_1):287-OR

https://doi.org/10.2337/db23-287-OR

Published: 20 June 2023

View article titled, 287-OR: An ABCC8 R1420H Loss-of-Function Variant Affects Insulin Secretory Function and Microvascular Disease in American Indians

Meeting Abstracts

286-OR: Using iPSC-Derived Islets to Model the Effect of an ABCC8 Arg1420His Mutation in Fetal and Adult Islets

ANUP K. NAIR, KATIYA BARKHO, MICHAEL TRAURIG, ROBERT G. NELSON, CLIFTON BOGARDUS, III, LESLIE BAIER

Diabetes 2023;72(Supplement_1):286-OR

https://doi.org/10.2337/db23-286-OR

Published: 20 June 2023

View article titled, 286-OR: Using iPSC-Derived Islets to Model the Effect of an ABCC8 Arg1420His Mutation in Fetal and Adult Islets

Meeting Abstracts

1274-P: Prevalence and Clinical Characteristics of Monogenic Diabetes in Chinese Population

SANDRA TSOI, CADMON K. LIM, RONALD C. MA, ERIC S. LAU, BAOQI FAN, ELAINE CHOW, ALICE P. KONG, JULIANA C. CHAN, ANDREA LUK

Diabetes 2022;71(Supplement_1):1274-P

https://doi.org/10.2337/db22-1274-P

Published: 01 June 2022

View article titled, 1274-P: Prevalence and Clinical Characteristics of Monogenic Diabetes in Chinese Population

Meeting Abstracts

301-OR: Modeling the Type 2 Diabetes–Associated ABCC8 Arg1420 His Mutation using iPSC–Derived Pancreatic Islet-Like Cells

ANUP K. NAIR, MICHAEL TRAURIG, JEFF SUTHERLAND, ROBERT G. NELSON, CLIFTON BOGARDUS, III, LESLIE BAIER

Diabetes 2022;71(Supplement_1):301-OR

https://doi.org/10.2337/db22-301-OR

Published: 01 June 2022

View article titled, 301-OR: Modeling the Type 2 Diabetes–Associated ABCC8 Arg1420 His Mutation using iPSC–Derived Pancreatic Islet-Like Cells

Journal Articles

Acyl-Ghrelin Influences Pancreatic β-Cell Function by Interference with K_ATP Channels

Julia Kaiser, Peter Krippeit-Drews, Gisela Drews

Diabetes 2021;70(2):423–435

https://doi.org/10.2337/db20-0231

Published: 05 November 2020

View article titled, Acyl-Ghrelin Influences Pancreatic β-Cell Function by Interference with KATP Channels

Meeting Abstracts

50-LB: Behavior and Learning Challenges in ABCC8-Related Neonatal Diabetes

NAIRI BROWN, SHAGUN BERRY, ANASTASIA HARRIS, LISA R. LETOURNEAU-FREIBERG, SIRI ATMA W. GREELEY

Diabetes 2020;69(Supplement_1):50-LB

https://doi.org/10.2337/db20-50-LB

Published: 01 June 2020

View article titled, 50-LB: Behavior and Learning Challenges in ABCC8-Related Neonatal Diabetes

Journal Articles

Systematic Genetic Study of Youth With Diabetes in a Single Country Reveals the Prevalence of Diabetes Subtypes, Novel Candidate Genes, and Response to Precision Therapy

Ingrida Stankute, Rasa Verkauskiene, Jean-Louis Blouin, Philippe Klee, Rimante Dobrovolskiene, Evalda Danyte, Mirjam Dirlewanger, Federico Santoni, Dovile Razanskaite-Virbickiene, Dale Marciulionyte, Edita Jasinskiene, Giedre Mockeviciene, Valerie M. Schwitzgebel

Diabetes 2020;69(5):1065–1071

https://doi.org/10.2337/db19-0974

Published: 21 February 2020

View article titled, Systematic Genetic Study of Youth With Diabetes in a Single Country Reveals the Prevalence of Diabetes Subtypes, Novel Candidate Genes, and Response to Precision Therapy

Includes: Supplementary data

Meeting Abstracts

142-LB: First Non-sulfonylurea Type Inhibitors of SUR1 Showed Superior Efficacy over Sulfonylureas, and Reversed Sulfonylurea Failure

DONGCHUL LIM, JUNG GYU PARK, DONG WOOK KIM, HEE DONG PARK, YOUNG KWAN KIM

Diabetes 2019;68(Supplement_1):142-LB

https://doi.org/10.2337/db19-142-LB

Published: 01 June 2019

View article titled, 142-LB: First Non-sulfonylurea Type Inhibitors of SUR1 Showed Superior Efficacy over Sulfonylureas, and Reversed Sulfonylurea Failure

Journal Articles

Chronic β-Cell Depolarization Impairs β-Cell Identity by Disrupting a Network of Ca²⁺-Regulated Genes

Jennifer S. Stancill, Jean-Philippe Cartailler, Hannah W. Clayton, James T. O’Connor, Matthew T. Dickerson, Prasanna K. Dadi, Anna B. Osipovich, David A. Jacobson, Mark A. Magnuson

Diabetes 2017;66(8):2175–2187

https://doi.org/10.2337/db16-1355

Published: 26 May 2017

View article titled, Chronic β-Cell Depolarization Impairs β-Cell Identity by Disrupting a Network of Ca2+-Regulated Genes

Includes: Supplementary data

Journal Articles

Genetic Variation at the Sulfonylurea Receptor, Type 2 Diabetes, and Coronary Heart Disease

Connor A. Emdin, Derek Klarin, Pradeep Natarajan, CARDIOGRAM Exome Consortium, Jose C. Florez, Sekar Kathiresan, Amit V. Khera

Diabetes 2017;66(8):2310–2315

https://doi.org/10.2337/db17-0149

Published: 14 April 2017

View article titled, Genetic Variation at the Sulfonylurea Receptor, Type 2 Diabetes, and Coronary Heart Disease

Includes: Supplementary data

Journal Articles

ABCC8 R1420H Loss-of-Function Variant in a Southwest American Indian Community: Association With Increased Birth Weight and Doubled Risk of Type 2 Diabetes

Leslie J. Baier, Yunhua Li Muller, Maria Sara Remedi, Michael Traurig, Paolo Piaggi, Gregory Wiessner, Ke Huang, Alyssa Stacy, Sayuko Kobes, Jonathan Krakoff, Peter H. Bennett, Robert G. Nelson, William C. Knowler, Robert L. Hanson, Colin G. Nichols, Clifton Bogardus

Diabetes 2015;64(12):4322–4332

https://doi.org/10.2337/db15-0459

Published: 05 August 2015

View article titled, ABCC8 R1420H Loss-of-Function Variant in a Southwest American Indian Community: Association With Increased Birth Weight and Doubled Risk of Type 2 Diabetes

Includes: Supplementary data

Journal Articles

Molecular Mechanism of Sulphonylurea Block of K_ATP Channels Carrying Mutations That Impair ATP Inhibition and Cause Neonatal Diabetes

Peter Proks, Heidi de Wet, Frances M. Ashcroft

Diabetes 2013;62(11):3909–3919

https://doi.org/10.2337/db13-0531

Published: 18 October 2013

View article titled, Molecular Mechanism of Sulphonylurea Block of KATP Channels Carrying Mutations That Impair ATP Inhibition and Cause Neonatal Diabetes

Journal Articles

A Mouse Model of Human Hyperinsulinism Produced by the E1506K Mutation in the Sulphonylurea Receptor SUR1

Kenju Shimomura, Maija Tusa, Michaela Iberl, Melissa F. Brereton, Stephan Kaizik, Peter Proks, Carolina Lahmann, Nagendra Yaluri, Shalem Modi, Hanna Huopio, Jarkko Ustinov, Timo Otonkoski, Markku Laakso, Frances M. Ashcroft

Diabetes 2013;62(11):3797–3806

https://doi.org/10.2337/db12-1611

Published: 18 October 2013

View article titled, A Mouse Model of Human Hyperinsulinism Produced by the E1506K Mutation in the Sulphonylurea Receptor SUR1

Includes: Supplementary data

Journal Articles

Relative Expression of a Dominant Mutated ABCC8 Allele Determines the Clinical Manifestation of Congenital Hyperinsulinism

Ruth Shemer, Carmit Avnon Ziv, Efrat Laiba, Qing Zhou, Joel Gay, Sharona Tunovsky-Babaey, Show-Ling Shyng, Benjamin Glaser, David H. Zangen

Diabetes 2012;61(1):258–263

https://doi.org/10.2337/db11-0984

Published: 12 December 2011

View article titled, Relative Expression of a Dominant Mutated ABCC8 Allele Determines the Clinical Manifestation of Congenital Hyperinsulinism

Includes: Supplementary data

Journal Articles

The ATP-Sensitive K⁺ Channel ABCC8 S1369A Type 2 Diabetes Risk Variant Increases MgATPase Activity

Mohammad Fatehi, Mobeen Raja, Christian Carter, Daniel Soliman, Andrew Holt, Peter E. Light

Diabetes 2012;61(1):241–249

https://doi.org/10.2337/db11-0371

Published: 12 December 2011

View article titled, The ATP-Sensitive K+ Channel ABCC8 S1369A Type 2 Diabetes Risk Variant Increases MgATPase Activity

Journal Articles

Erratum

Diabetes 2011;60(11):3097

https://doi.org/10.2337/db11-er11

Published: 17 October 2011

View article titled, Erratum

Journal Articles

Diazoxide-Unresponsive Congenital Hyperinsulinism in Children With Dominant Mutations of the β-Cell Sulfonylurea Receptor SUR1

Courtney M. MacMullen, Qing Zhou, Kara E. Snider, Paul H. Tewson, Susan A. Becker, Ali Rahim Aziz, Arupa Ganguly, Show-Ling Shyng, Charles A. Stanley

Diabetes 2011;60(6):1797–1804

https://doi.org/10.2337/db10-1631

Published: 21 May 2011

View article titled, Diazoxide-Unresponsive Congenital Hyperinsulinism in Children With Dominant Mutations of the β-Cell Sulfonylurea Receptor SUR1

Journal Articles

Mutations of the Same Conserved Glutamate Residue in NBD2 of the Sulfonylurea Receptor 1 Subunit of the K_ATP Channel Can Result in Either Hyperinsulinism or Neonatal Diabetes

Roope Männikkö, Sarah E. Flanagan, Xiuli Sim, David Segal, Khalid Hussain, Sian Ellard, Andrew T. Hattersley, Frances M. Ashcroft

Diabetes 2011;60(6):1813–1822

https://doi.org/10.2337/db10-1583

Published: 21 May 2011

View article titled, Mutations of the Same Conserved Glutamate Residue in NBD2 of the Sulfonylurea Receptor 1 Subunit of the KATP Channel Can Result in Either Hyperinsulinism or Neonatal Diabetes

Includes: Supplementary data

NARROW

Loss of ATP-Sensitive Potassium Channel Expression and Function in the Nervous System Decreases Opioid Sensitivity in a High-Fat Diet–Fed Mouse Model of Diet-Induced Obesity

1749-P: Influence of the Biomechanical Environment on Islet Function and Maturity

287-OR: An ABCC8 R1420H Loss-of-Function Variant Affects Insulin Secretory Function and Microvascular Disease in American Indians

286-OR: Using iPSC-Derived Islets to Model the Effect of an ABCC8 Arg1420His Mutation in Fetal and Adult Islets

1274-P: Prevalence and Clinical Characteristics of Monogenic Diabetes in Chinese Population

301-OR: Modeling the Type 2 Diabetes–Associated ABCC8 Arg1420 His Mutation using iPSC–Derived Pancreatic Islet-Like Cells

Acyl-Ghrelin Influences Pancreatic β-Cell Function by Interference with K_ATP Channels

50-LB: Behavior and Learning Challenges in ABCC8-Related Neonatal Diabetes

Systematic Genetic Study of Youth With Diabetes in a Single Country Reveals the Prevalence of Diabetes Subtypes, Novel Candidate Genes, and Response to Precision Therapy

142-LB: First Non-sulfonylurea Type Inhibitors of SUR1 Showed Superior Efficacy over Sulfonylureas, and Reversed Sulfonylurea Failure

Chronic β-Cell Depolarization Impairs β-Cell Identity by Disrupting a Network of Ca²⁺-Regulated Genes

Genetic Variation at the Sulfonylurea Receptor, Type 2 Diabetes, and Coronary Heart Disease

ABCC8 R1420H Loss-of-Function Variant in a Southwest American Indian Community: Association With Increased Birth Weight and Doubled Risk of Type 2 Diabetes

Molecular Mechanism of Sulphonylurea Block of K_ATP Channels Carrying Mutations That Impair ATP Inhibition and Cause Neonatal Diabetes

A Mouse Model of Human Hyperinsulinism Produced by the E1506K Mutation in the Sulphonylurea Receptor SUR1

Relative Expression of a Dominant Mutated ABCC8 Allele Determines the Clinical Manifestation of Congenital Hyperinsulinism

The ATP-Sensitive K⁺ Channel ABCC8 S1369A Type 2 Diabetes Risk Variant Increases MgATPase Activity

Erratum

Diazoxide-Unresponsive Congenital Hyperinsulinism in Children With Dominant Mutations of the β-Cell Sulfonylurea Receptor SUR1

Mutations of the Same Conserved Glutamate Residue in NBD2 of the Sulfonylurea Receptor 1 Subunit of the K_ATP Channel Can Result in Either Hyperinsulinism or Neonatal Diabetes

Email alerts

Journals

Books

Clinical Compendia

News

Other

About

Resources

Email alerts

Journals

Books

Clinical Compendia

News

Other

About

Resources

This Feature Is Available To Subscribers Only