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Journal Articles

Central K_ATP Channels Modulate Glucose Effectiveness in Humans and Rodents

Michelle Carey, Eric Lontchi-Yimagou, William Mitchell, Sarah Reda, Kehao Zhang, Sylvia Kehlenbrink, Sudha Koppaka, Sylvan Roger Maginley, Sandra Aleksic, Shobhit Bhansali, Derek M. Huffman, Meredith Hawkins

Diabetes 2020;69(6):1140–1148

DOI: https://doi.org/10.2337/db19-1256

Published: 26 March 2020

View article titled, Central KATP Channels Modulate Glucose Effectiveness in Humans and Rodents

Journal Articles

Cerebrovascular Safety of Sulfonylureas: The Role of K_ATP Channels in Neuroprotection and the Risk of Stroke in Patients With Type 2 Diabetes

Rui Liu, Haitao Wang, Baofeng Xu, Wenliang Chen, Ekaterina Turlova, Nan Dong, Christopher L.F. Sun, Yangqingqin Lu, Hanhui Fu, Ranran Shi, Andrew Barszczyk, Dongzi Yang, Tianru Jin, Edoardo Mannucci, Zhong-Ping Feng, Hong-Shuo Sun

Diabetes 2016;65(9):2795–2809

DOI: https://doi.org/10.2337/db15-1737

Published: 13 May 2016

View article titled, Cerebrovascular Safety of Sulfonylureas: The Role of KATP Channels in Neuroprotection and the Risk of Stroke in Patients With Type 2 Diabetes

Journal Articles

Central Regulation of Glucose Production May Be Impaired in Type 2 Diabetes

Yonah B. Esterson, Michelle Carey, Laura Boucai, Akankasha Goyal, Pooja Raghavan, Kehao Zhang, Deeksha Mehta, Daorong Feng, Licheng Wu, Sylvia Kehlenbrink, Sudha Koppaka, Preeti Kishore, Meredith Hawkins

Diabetes 2016;65(9):2569–2579

DOI: https://doi.org/10.2337/db15-1465

Published: 10 May 2016

View article titled, Central Regulation of Glucose Production May Be Impaired in Type 2 Diabetes

Includes: Supplementary data

Journal Articles

Human Insulinomas Show Distinct Patterns of Insulin Secretion In Vitro

Jean-Claude Henquin, Myriam Nenquin, Yves Guiot, Jacques Rahier, Christine Sempoux

Diabetes 2015;64(10):3543–3553

DOI: https://doi.org/10.2337/db15-0527

Published: 26 June 2015

View article titled, Human Insulinomas Show Distinct Patterns of Insulin Secretion In Vitro

Includes: Supplementary data

Journal Articles

Diazoxide Improves Hormonal Counterregulatory Responses to Acute Hypoglycemia in Long-standing Type 1 Diabetes

Priya S. George, Roger Tavendale, Colin N.A. Palmer, Rory J. McCrimmon

Diabetes 2015;64(6):2234–2241

DOI: https://doi.org/10.2337/db14-1539

Published: 15 January 2015

View article titled, Diazoxide Improves Hormonal Counterregulatory Responses to Acute Hypoglycemia in Long-standing Type 1 Diabetes

Includes: Supplementary data

Journal Articles

Congenital Hyperinsulinism Caused by Hexokinase I Expression or Glucokinase-Activating Mutation in a Subset of β-Cells

Jean-Claude Henquin, Christine Sempoux, Joelle Marchandise, Sebastien Godecharles, Yves Guiot, Myriam Nenquin, Jacques Rahier

Diabetes 2013;62(5):1689–1696

DOI: https://doi.org/10.2337/db12-1414

Published: 16 April 2013

View article titled, Congenital Hyperinsulinism Caused by Hexokinase I Expression or Glucokinase-Activating Mutation in a Subset of β-Cells

Includes: Supplementary data

Journal Articles

New Insights and New Conundrums in Neonatal Hypoglycemia: Enigmas Wrapped in Mystery

Mark A. Sperling

Diabetes 2013;62(5):1373–1375

DOI: https://doi.org/10.2337/db12-1839

Published: 16 April 2013

View article titled, New Insights and New Conundrums in Neonatal Hypoglycemia: Enigmas Wrapped in Mystery

Journal Articles

Erratum

Diabetes 2011;60(11):3097

DOI: https://doi.org/10.2337/db11-er11

Published: 17 October 2011

View article titled, Erratum

Journal Articles

Diazoxide-Unresponsive Congenital Hyperinsulinism in Children With Dominant Mutations of the β-Cell Sulfonylurea Receptor SUR1

Courtney M. MacMullen, Qing Zhou, Kara E. Snider, Paul H. Tewson, Susan A. Becker, Ali Rahim Aziz, Arupa Ganguly, Show-Ling Shyng, Charles A. Stanley

Diabetes 2011;60(6):1797–1804

DOI: https://doi.org/10.2337/db10-1631

Published: 21 May 2011

View article titled, Diazoxide-Unresponsive Congenital Hyperinsulinism in Children With Dominant Mutations of the β-Cell Sulfonylurea Receptor SUR1

Journal Articles

In Vitro Recovery of ATP-Sensitive Potassium Channels in β-Cells From Patients With Congenital Hyperinsulinism of Infancy

Philippa D. Powell, Christine Bellanné-Chantelot, Sarah E. Flanagan, Sian Ellard, Raoul Rooman, Khalid Hussain, Mars Skae, Peter Clayton, Pascale de Lonlay, Mark J. Dunne, Karen E. Cosgrove

Diabetes 2011;60(4):1223–1228

DOI: https://doi.org/10.2337/db10-1443

Published: 22 March 2011

View article titled, In Vitro Recovery of ATP-Sensitive Potassium Channels in β-Cells From Patients With Congenital Hyperinsulinism of Infancy

Journal Articles

Partial Inhibition of Insulin Secretion Results in Glucose Intolerance but Not Hyperglucagonemia

Ranjani P. Ramanathan, Ana María Arbeláez, Philip E. Cryer

Diabetes 2011;60(4):1324–1328

DOI: https://doi.org/10.2337/db10-1586

Published: 22 March 2011

View article titled, Partial Inhibition of Insulin Secretion Results in Glucose Intolerance but Not Hyperglucagonemia

Journal Articles

Congenital Hyperinsulinism and Glucose Hypersensitivity in Homozygous and Heterozygous Carriers of Kir6.2 (KCNJ11) Mutation V290M Mutation: K_ATP Channel Inactivation Mechanism and Clinical Management

Karen J. Loechner, Alejandro Akrouh, Harley T. Kurata, Carlo Dionisi-Vici, Arianna Maiorana, Milena Pizzoferro, Vittoria Rufini, Jean de Ville de Goyet, Carlo Colombo, Fabrizio Barbetti, Joseph C. Koster, Colin G. Nichols

Diabetes 2011;60(1):209–217

DOI: https://doi.org/10.2337/db10-0731

Published: 27 October 2010

View article titled, Congenital Hyperinsulinism and Glucose Hypersensitivity in Homozygous and Heterozygous Carriers of Kir6.2 (KCNJ11) Mutation V290M Mutation: KATP Channel Inactivation Mechanism and Clinical Management

Journal Articles

ATP-Sensitive K⁺ Channel Mediates the Zinc Switch-Off Signal for Glucagon Response During Glucose Deprivation

Michela Slucca, Jamie S. Harmon, Elizabeth A. Oseid, Joseph Bryan, R. Paul Robertson

Diabetes 2010;59(1):128–134

DOI: https://doi.org/10.2337/db09-1098

Published: 06 October 2009

View article titled, ATP-Sensitive K+ Channel Mediates the Zinc Switch-Off Signal for Glucagon Response During Glucose Deprivation

Journal Articles

Extremes of Clinical and Enzymatic Phenotypes in Children With Hyperinsulinism Caused by Glucokinase Activating Mutations

Samir Sayed, David R. Langdon, Stella Odili, Pan Chen, Carol Buettger, Alisa B. Schiffman, Mariko Suchi, Rebecca Taub, Joseph Grimsby, Franz M. Matschinsky, Charles A. Stanley

Diabetes 2009;58(6):1419–1427

DOI: https://doi.org/10.2337/db08-1792

Published: 31 March 2009

View article titled, Extremes of Clinical and Enzymatic Phenotypes in Children With Hyperinsulinism Caused by Glucokinase Activating Mutations

Journal Articles

Persistent Hyperinsulinemic Hypoglycemia and Maturity-Onset Diabetes of the Young Due to Heterozygous HNF4A Mutations

Ritika R. Kapoor, Jonathan Locke, Kevin Colclough, Jerry Wales, Jennifer J. Conn, Andrew T. Hattersley, Sian Ellard, Khalid Hussain

Diabetes 2008;57(6):1659–1663

DOI: https://doi.org/10.2337/db07-1657

Published: 01 June 2008

View article titled, Persistent Hyperinsulinemic Hypoglycemia and Maturity-Onset Diabetes of the Young Due to Heterozygous HNF4A Mutations

Journal Articles

Acute Diabetes Moderates Trafficking of Cardiac Lipoprotein Lipase Through p38 Mitogen-Activated Protein Kinase–Dependent Actin Cytoskeleton Organization

Min Suk Kim, Girish Kewalramani, Prasanth Puthanveetil, Vivian Lee, Ujendra Kumar, Ding An, Ashraf Abrahani, Brian Rodrigues

Diabetes 2008;57(1):64–76

DOI: https://doi.org/10.2337/db07-0832

Published: 01 January 2008

View article titled, Acute Diabetes Moderates Trafficking of Cardiac Lipoprotein Lipase Through p38 Mitogen-Activated Protein Kinase–Dependent Actin Cytoskeleton Organization

Journal Articles

Congenital Hyperinsulinism–Associated ABCC8 Mutations That Cause Defective Trafficking of ATP-Sensitive K⁺ Channels: Identification and Rescue

Fei-Fei Yan, Yu-Wen Lin, Courtney MacMullen, Arupa Ganguly, Charles A. Stanley, Show-Ling Shyng

Diabetes 2007;56(9):2339–2348

DOI: https://doi.org/10.2337/db07-0150

Published: 01 September 2007

View article titled, Congenital Hyperinsulinism–Associated ABCC8 Mutations That Cause Defective Trafficking of ATP-Sensitive K+ Channels: Identification and Rescue

Includes: Supplementary data

Journal Articles

The Actions of a Novel Potent Islet β-Cell–Specific ATP-Sensitive K⁺ Channel Opener Can Be Modulated by Syntaxin-1A Acting on Sulfonylurea Receptor 1

Betty Ng, Youhou Kang, Chadwick L. Elias, Yan He, Huanli Xie, John B. Hansen, Philip Wahl, Herbert Y. Gaisano

Diabetes 2007;56(8):2124–2134

DOI: https://doi.org/10.2337/db07-0030

Published: 01 August 2007

View article titled, The Actions of a Novel Potent Islet β-Cell–Specific ATP-Sensitive K+ Channel Opener Can Be Modulated by Syntaxin-1A Acting on Sulfonylurea Receptor 1

Includes: Supplementary data

Journal Articles

ATP-Sensitive K⁺ Channels Regulate the Release of GABA in the Ventromedial Hypothalamus During Hypoglycemia

Owen Chan, Marcus Lawson, Wanling Zhu, Joseph Lee Beverly, Robert S. Sherwin

Diabetes 2007;56(4):1120–1126

DOI: https://doi.org/10.2337/db06-1102

Published: 01 April 2007

View article titled, ATP-Sensitive K+ Channels Regulate the Release of GABA in the Ventromedial Hypothalamus During Hypoglycemia

Journal Articles

Effects of Diazoxide on Gene Expression in Rat Pancreatic Islets Are Largely Linked to Elevated Glucose and Potentially Serve to Enhance β-Cell Sensitivity

Zuheng Ma, Neil Portwood, David Brodin, Valdemar Grill, Anneli Björklund

Diabetes 2007;56(4):1095–1106

DOI: https://doi.org/10.2337/db06-0322

Published: 01 April 2007

View article titled, Effects of Diazoxide on Gene Expression in Rat Pancreatic Islets Are Largely Linked to Elevated Glucose and Potentially Serve to Enhance β-Cell Sensitivity

Includes: Supplementary data

NARROW

Central K_ATP Channels Modulate Glucose Effectiveness in Humans and Rodents

Cerebrovascular Safety of Sulfonylureas: The Role of K_ATP Channels in Neuroprotection and the Risk of Stroke in Patients With Type 2 Diabetes

Central Regulation of Glucose Production May Be Impaired in Type 2 Diabetes

Human Insulinomas Show Distinct Patterns of Insulin Secretion In Vitro

Diazoxide Improves Hormonal Counterregulatory Responses to Acute Hypoglycemia in Long-standing Type 1 Diabetes

Congenital Hyperinsulinism Caused by Hexokinase I Expression or Glucokinase-Activating Mutation in a Subset of β-Cells

New Insights and New Conundrums in Neonatal Hypoglycemia: Enigmas Wrapped in Mystery

Erratum

Diazoxide-Unresponsive Congenital Hyperinsulinism in Children With Dominant Mutations of the β-Cell Sulfonylurea Receptor SUR1

In Vitro Recovery of ATP-Sensitive Potassium Channels in β-Cells From Patients With Congenital Hyperinsulinism of Infancy

Partial Inhibition of Insulin Secretion Results in Glucose Intolerance but Not Hyperglucagonemia

Congenital Hyperinsulinism and Glucose Hypersensitivity in Homozygous and Heterozygous Carriers of Kir6.2 (KCNJ11) Mutation V290M Mutation: K_ATP Channel Inactivation Mechanism and Clinical Management

ATP-Sensitive K⁺ Channel Mediates the Zinc Switch-Off Signal for Glucagon Response During Glucose Deprivation

Extremes of Clinical and Enzymatic Phenotypes in Children With Hyperinsulinism Caused by Glucokinase Activating Mutations

Persistent Hyperinsulinemic Hypoglycemia and Maturity-Onset Diabetes of the Young Due to Heterozygous HNF4A Mutations

Acute Diabetes Moderates Trafficking of Cardiac Lipoprotein Lipase Through p38 Mitogen-Activated Protein Kinase–Dependent Actin Cytoskeleton Organization

Congenital Hyperinsulinism–Associated ABCC8 Mutations That Cause Defective Trafficking of ATP-Sensitive K⁺ Channels: Identification and Rescue

The Actions of a Novel Potent Islet β-Cell–Specific ATP-Sensitive K⁺ Channel Opener Can Be Modulated by Syntaxin-1A Acting on Sulfonylurea Receptor 1

ATP-Sensitive K⁺ Channels Regulate the Release of GABA in the Ventromedial Hypothalamus During Hypoglycemia

Effects of Diazoxide on Gene Expression in Rat Pancreatic Islets Are Largely Linked to Elevated Glucose and Potentially Serve to Enhance β-Cell Sensitivity

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