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Meeting Abstracts

1756-P: Extracellular Matrix Stiffness Mediates Insulin Secretion in Pancreatic Islets via Phosphofructokinase Activity

CHELSEA GARCIA, KEIFER HOLCOMB, NIKKI L. FARNSWORTH
Journal: Diabetes
Diabetes 2023;72(Supplement_1):1756-P
https://doi.org/10.2337/db23-1756-P
Published: 20 June 2023
View article titled, 1756-P: Extracellular Matrix Stiffness Mediates Insulin Secretion in Pancreatic Islets via Phosphofructokinase Activity
Journal Articles

Expression of Phosphofructokinase in Skeletal Muscle Is Influenced by Genetic Variation and Associated With Insulin Sensitivity

Sarah Keildson, Joao Fadista, Claes Ladenvall, Åsa K. Hedman, Targ Elgzyri, Kerrin S. Small, Elin Grundberg, Alexandra C. Nica, Daniel Glass, J. Brent Richards, Amy Barrett, James Nisbet, Hou-Feng Zheng, Tina Rönn, Kristoffer Ström, Karl-Fredrik Eriksson, Inga Prokopenko, MAGIC Consortium, DIAGRAM Consortium, MuTHER Consortium, Timothy D. Spector, Emmanouil T. Dermitzakis, Panos Deloukas, Mark I. McCarthy, Johan Rung, Leif Groop, Paul W. Franks, Cecilia M. Lindgren, Ola Hansson
Journal: Diabetes
Diabetes 2014;63(3):1154–1165
https://doi.org/10.2337/db13-1301
Published: 13 February 2014
View article titled, Expression of Phosphofructokinase in Skeletal Muscle Is Influenced by Genetic Variation and Associated With Insulin Sensitivity
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Includes: Supplementary data
Meeting Abstracts

Shared biochemical properties of glucotoxicity and lipotoxicity in islets decrease citrate synthase activity and increase phosphofructokinase activity.

Y Q Liu, K Tornheim, J L Leahy
Journal: Diabetes
Diabetes 1998;47(12):1889–1893
https://doi.org/10.2337/diabetes.47.12.1889
Published: 01 December 1998
View article titled, Shared biochemical properties of glucotoxicity and lipotoxicity in islets decrease citrate synthase activity and increase phosphofructokinase activity.
Open the PDF for Shared biochemical properties of glucotoxicity and lipotoxicity in islets decrease citrate synthase activity and increase phosphofructokinase activity. in another window
Journal Articles

Impaired Glucose-Induced Insulin Response in Transgenic Mice Overexpressing the L-phosphofructokinase Gene

Hilla Knobler, Yael Weiss, Mira Peled, Yoram Groner
Journal: Diabetes
Diabetes 1997;46(9):1414–1418
https://doi.org/10.2337/diab.46.9.1414
Published: 01 September 1997
View article titled, Impaired Glucose-Induced Insulin Response in Transgenic Mice Overexpressing the L-phosphofructokinase Gene
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Journal Articles

Are Metabolic Oscillations Responsible for Normal Oscillatory Insulin Secretion?

Keith Tornheim
Journal: Diabetes
Diabetes 1997;46(9):1375–1380
https://doi.org/10.2337/diab.46.9.1375
Published: 01 September 1997
View article titled, Are Metabolic Oscillations Responsible for Normal Oscillatory Insulin Secretion?
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Journal Articles

Phosphofructokinase Isozymes in Pancreatic Islets and Clonal β-Cells (INS-1)

Gordon C Yaney, Vera Schultz, Barbara A Cunningham, George A Dunaway, Barbara E Corkey, Keith Tornheim
Journal: Diabetes
Diabetes 1995;44(11):1285–1289
https://doi.org/10.2337/diab.44.11.1285
Published: 01 November 1995
View article titled, Phosphofructokinase Isozymes in Pancreatic Islets and Clonal β-Cells (INS-1)
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Journal Articles

Control of Glucose Metabolism in Pancreatic β-Cells by Glucokinase, Hexokinase and Phosphofructokinase: Model Study With Cell Lines Derived From β-Cells

Takao Shimizu, Janice C Parker, Habiba Najafi, Franz M Matschinsky
Journal: Diabetes
Diabetes 1988;37(11):1524–1530
https://doi.org/10.2337/diab.37.11.1524
Published: 01 November 1988
View article titled, Control of Glucose Metabolism in Pancreatic β-Cells by Glucokinase, Hexokinase and Phosphofructokinase: Model Study With Cell Lines Derived From β-Cells
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Journal Articles

Postreceptor Myocardial Metabolic Defect in a Rat Model of Non-Insulin-Dependent Diabetes Mellitus

Stephen W Schaffer, Mahmood Seyed-Mozaffari, Cathleen R Cutcliff, Glenn L Wilson
Journal: Diabetes
Diabetes 1986;35(5):593–597
https://doi.org/10.2337/diab.35.5.593
Published: 01 May 1986
View article titled, Postreceptor Myocardial Metabolic Defect in a Rat Model of Non-Insulin-Dependent Diabetes Mellitus
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